Vitamin D
International Encyclopedia of Public Health (Second Edition) 2017, Pages 383–387; http://dx.doi.org/10.1016/B978-0-12-803678-5.00488-4
Reinhold Vieth
Blurry image from the free abstract
See also Vitamin D Life
- Vieth is on 828 pages as of Oct 2016
- Thoughts on improving vitamin D policies - Vieth Nov 2013
- Benefits and risks of Vitamin D - Vieth April 2012
Vitamin D Receptor category has the following
Vitamin D tests cannot detect Vitamin D Receptor (VDR) problems
A poor VDR restricts Vitamin D from getting in the cells
It appears that 30% of the population have a poor VDR (40% of the Obese )
A poor VDR increases the risk of 55 health problems click here for details
The risk of 44 diseases at least double with poor Vitamin D Receptor as of Oct 2019
VDR at-home test $29 - results not easily understood in 2016
There are hints that you may have inherited a poor VDR
Compensate for poor VDR by increasing one or more:
| Increasing | Increases |
| 1) Vitamin D supplement Sun, Ultraviolet -B | Vitamin D in the blood and thus in the cells |
| 2) Magnesium | Vitamin D in the blood AND in the cells |
| 3) Omega-3 | Vitamin D in the cells |
| 4) Resveratrol | Vitamin D Receptor |
| 5) Intense exercise | Vitamin D Receptor |
| 6) Get prescription for VDR activator paricalcitol, maxacalcitol? | Vitamin D Receptor |
| 7) Quercetin (flavonoid) | Vitamin D Receptor |
| 8) Zinc is in the VDR | Vitamin D Receptor |
| 9) Boron | Vitamin D Receptor ?, etc |
| 10) Essential oils e.g. ginger, curcumin | Vitamin D Receptor |
| 11) Progesterone | Vitamin D Receptor |
| 12) Infrequent high concentration Vitamin D Increases the concentration gradient | Vitamin D in the cells |
| 13) Sulfroaphone and perhaps sulfur | Vitamin D Receptor |
Note: If you are not feeling enough benefit from Vitamin D, you might try increasing VDR activation. You might feel the benefit within days of adding one or more of the above
Far healthier and stronger at age 72 due to supplements Includes 6 supplements which help the VDR
Vitamin D deficiency causes rickets in children and osteomalacia in adults, both of which are accompanied by muscle weakness. Deficiency is measureable by a blood test of the vitamin D metabolite, 25-hydroxyvitamin D [25(OH)D]. Disagreements about the need for vitamin D are based on differing opinions about desirable health-related thresholds for serum 25(OH)D.
Debates are also about how much extra vitamin D might be needed to sustain those thresholds and about whether health criteria in addition to the bone, muscle, pain, and falls are pertinent to establishing the desired 25(OH)D level. The Institutes of Medicine considers 25(OH)D levels below 50 nmol L-1 (20 ng mL-1) as undesirable, and in some countries, average ‘normal’ population levels are lower than that. Vitamin D is produced naturally in skin exposed to ultraviolet (UV)-B light.
Vitamin D is not produced in the skin covered by clothing or if the UV index is below 4
(i.e., if the angle of the sun is so low that a person's shadow is longer than his/her height).
Vitamin D is not naturally present in most foods, but public health advice to fortify food or to supplement with it remains controversial.
At latitudes south of 30 degrees, the most cost-effective approach is to recommend more exposure of the skin to sunshine, but that is complicated by local culture and worries about skin cancer.
Public health policies surrounding vitamin D and sun exposure remain unsettled.
Meanwhile, intense research interest in the field continues to produce provocative results.
Publisher wants $31.50 for the 4 pages
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