Could Vitamin D3 Deficiency Influence Malocclusion Development?
Nutrients 2021, 13(6), 2122; https://doi.org/10.3390/nu13062122
by Anna Leszczyszyn 1,Sylwia Hnitecka 2,*OrcID andMarzena Dominiak 1
1 Oral Surgery Department, Wroclaw Medical University, 50-425 Wroclaw, Poland
2 Maxillofacial Surgery Department, Wroclaw Medical University, 50-556 Wroclaw, Poland
Vitamin D Life - Dental category contains
Some Dental studies
- Vitamin D cut dental caries in half 80 years ago – meta-analysis 2013 80 years ago
- Severe periodontitis is half as likely at 60 ng of vitamin D – Sept 2021
- Risk of Oral Surgery reduced by Vitamin D – scoping review Nov 2021
- Some Dental Malocclusions 5X more likely if low vitamin D – June 2021
- Grinding teeth while sleeping (sleep bruxism) 6 X more likely if low vitamin D – Jan 2021
- Vitamin D Deficiency and Oral Health: A Comprehensive Review – May 2020
- 1.8 X more likely to have additional Dental Implant Failure if poor Vitamin D Receptor – Dec 2019
- Early tooth decay 1.9 X more likely if a poor Vitamin D receptor – July 2017
Wikipedia
In orthodontics, a malocclusion is a misalignment or incorrect relation between the teeth of the upper and lower dental arches when they approach each other as the jaws close.
 Download the PDF from Vitamin D Life
The abnormal growth of the craniofacial bone leads to skeletal and dental defects, which result in the presence of malocclusions. Not all causes of malocclusion have been explained. In the development of skeletal abnormalities, attention is paid to general deficiencies, including of vitamin D3 (VD3), which causes rickets. Its chronic deficiency may contribute to skeletal malocclusion. The aim of the study was to assess the impact of VD3 deficiency on the development of malocclusions. The examination consisted of a medical interview, oral examination, an alginate impression and radiological imaging, orthodontic assessment, and taking a venous blood sample for VD3 level testing. In about 42.1% of patients, the presence of a skeletal defect was found, and in 46.5% of patients, dentoalveolar malocclusion. The most common defect was transverse constriction of the maxilla with a narrow upper arch (30.7%). The concentration of vitamin 25 (OH) D in the study group was on average 23.6 ± 10.5 (ng/mL). VD3 deficiency was found in 86 subjects (75.4%). Our research showed that VD3 deficiency could be one of an important factor influencing maxillary development. Patients had a greater risk of a narrowed upper arch (OR = 4.94), crowding (OR = 4.94) and crossbite (OR = 6.16). Thus, there was a link between the deficiency of this hormone and the underdevelopment of the maxilla
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