Traumatic Brain Injury recovery helped by injection of 100,000 IU of Vitamin D

Created May 2019

The Effect of Vitamin D Supplementation in Patients with Acute Traumatic Brain Injury

World Neurosurgery, online 20 March 2019, https://doi.org/10.1016/j.wneu.2019.02.244

This study used just a small injection of Vitamin DSuspect they injected regular vitamin D into muscle or fat tissueExpect faster response from injecting semiactivated or fully activated Vitamin D into the blood100,000 IU of Vitamin D will raise the blood level a little bitLoading doses of 400,000 and 600,000 IU of Vitamin D have been used >1 million times     By the way - I take 100,000 IU of vitamin D every 4 days (founder of Vitamin D Life May 2019)* Mild Traumatic Brain Injury reduced by Vitamin D – May 2014* Vitamin D aided progesterone in reducing traumatic brain injury – RCT Dec 2012* Mild Traumatic Brain Injury prevented with Omega-3, Resveratrol, etc (in rats) – Oct 2017* Football Brain injuries prevented by Omega-3 – RCT Jan 2016* Magnesium may be an important way to treat brain trauma* Vitamin D and Glutamine reduced Trauma Center deaths by half – Matthews March 2017* Vitamin D, trauma, and blacks - Dr. Matthews interview with transcript - Jan 2019* Vitamin D3 Attenuates Blood-Brain Barrier Disruption due to TBI (in rats) – Feb 2021--- 1. TBI appears to be prevented and/or treated by getting more vitamin D in the brainEach of the following help, some combination should be really good1. Progesterone (VDR)1. Omega-3 (VDR)1. Resveratrol (VDR)1. Magnesium (VDR and blood)1. Vitamin DWonder which additonal VDR activators might also prevent/treat TBI 1. Vitamin D Receptor table shows what compensates for low VDR activation{include}The risk of 40 diseases at least double with poor Vitamin D Receptor as of July 2019--- 1. Trauma and surgery category starts with the following{include}

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Objective: To investigate the acute and long-term effects of vitamin D supplementation on the recovery of patients with traumatic brain injury (TBI).

Methods

A retrospective study was conducted involving 345 patients with TBI who visited a single trauma center. Vitamin D serum levels were measured without supplementation at admission, 1 month, and 3 months post-TBI (control group) from August to December 2016. From January 2017, vitamin D supplementation was provided to patients with TBI with low vitamin D serum levels at admission (supplement group). The outcomes were investigated by assessing performance function (Extended Glasgow Outcome Scale) and cognitive function (Mini-Mental Status Examination, and Clinical Dementia Rating) at 1 week and 3 months post-TBI.

Results

The mean vitamin D serum level in patients with TBI at admission was 13.62 ± 9.01 ng/mL. The level significantly increased from 14.03 ± 8.68 ng/mL at admission to 37.42 ± 12.57 ng/mL at 3 months post TBI in the supplement group (P < 0.001). The cognitive outcomes (Mini-Mental Status Examination/Clinical Dementia Rating, P = 0.042/P = 0.044) and GOS-E score (total TBI, P = 0.003; mild-to-moderate TBI, P = 0.002) significantly improved from the first week to 3 months post TBI in the patients with vitamin D supplementation.

Conclusions

Administration of vitamin D supplements in mild-to-moderate TBI patients with significant vitamin D deficiency during the acute phase of the injury may improve long-term performance and cognitive outcomes. Therefore, the treatment strategies should be individually planned for the patients with TBI based on their baseline vitamin D level.

Tags: Injection Loading dose Trauma and surgery Vit D Receptor